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It has been demonstrated that oxidative stress-mediated GSH depletion is crucial in controlling transient outward K+ currents in rat ventricular myocytes, which may be the cause of diabetes-related decreased contractility and aberrant electrical characteristics

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S-Acetyl L-Glutathione rapidly elevates cellular glutathione concentrations, making it particularly valuable during periods of increased physical or mental stress when the body's glutathione reserves are being utilized more quickly than usual